Why lowering estrogen may help fix all health problems

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How to tell if you have high estrogen levels and what to do about it if you do (most men have high levels)…

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New video reveals simple solo activities that work better than the “little blue pill”

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Why lowering estrogen may help fix all health problems for men

Estrogen is known as a female hormone.

While it is true that pre-menopausal women tend to have more estrogen than men, many men have HIGHER estrogen levels than their menopausal wives.

And in fact, estrogen is really not a sex hormone.

Estrogen might be better understood as an inflammatory hormone.

High levels of estrogen are linked to increased rates of disease in both men and women.

In recent years, researchers have tied high estrogen levels to a number of “autoimmune diseases.” 

These are diseases where the body seems to attack itself for no reason.

A recent study found another incriminating link between estrogen and autoimmune disease.

The study found that men with systemic scleroderma were more likely to also have high estrogen. 

High estrogen levels were also linked to more severe progression of the disease.

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These scientists did their research at the Medical University of South Carolina, Charleston, SC, USA. Arthritis Research & Therapy published the results.

Scleroderma is a disease of the connective tissue. 

The more extreme form, systemic scleroderma, causes an accumulation of fibrous collagen protein.

In systemic scleroderma, this excess protein can cause serious damage to all of the major organs.

Like many autoimmune diseases, it affects more women than men.

“Systemic scleroderma a female-predominant disease, characterized by excessive extracellular matrix deposition with dermal and internal organ fibrosis…”

Researchers became suspicious about the role of estrogen in systemic scleroderma because of the sex differences in risk.

“Considering the sex-based disparity in disease incidence, estrogen with pro-fibrotic effects may play a role in systemic scleroderma.”

The researchers recruited men over the age of 50 who had a diagnosis of systemic scleroderma for less than two years.

The study also enrolled healthy men in the same age group.

The researchers took blood samples and looked at one of the most common forms of estrogen – estradiol (E2).

“We measured serum estrogen E2 in 83 sclerodermic men over 50 years old – and similar aged healthy controls.”

The researchers looked at the relationship between estrogen and important factors associated with systemic scleroderma.

“We examined the associations between estrogen E2, internal organ involvement, autoantibody profiles, and survival.”

Men with systemic scleroderma had much higher estrogen levels than the healthy men in the same age group.

“Male systemic scleroderma patients had significantly higher estrogen levels compared to healthy males.”

Men with a diagnosis of systemic scleroderma even had higher levels of estrogen than women with the disease!

“Male systemic scleroderma patients had significantly higher estrogen levels compared to post-menopausal female patients.”

Estrogen levels predicted heart problems associated with the disease.

Men with higher estrogen levels were more likely to have systemic scleroderma affecting the heart.

Estrogen levels were almost 50% higher in men who had heart problems attributed to systemic scleroderma.

“Male patients with systemic scleroderma and heart involvement had significantly higher estrogen levels to those without heart involvement.”

Changes in skin thickness are one of the earliest and most obvious signs of systemic scleroderma progression.

Men with higher levels of estrogen showed accelerated skin thickness progression.

Finally, estrogen levels were related to disease survival.

Men with lower levels of estrogen were more likely to survive longer after diagnosis.

“Male patients with high estrogen had significantly more heart involvement, higher skin thickness progression rate, and worse survival.”

Autoimmune diseases like systemic scleroderma are triggered by antibodies. 

These are proteins produced by the body which attack and damage the body itself.

Men with higher levels of estrogen and positive systemic scleroderma antibodies were more likely to die from the disease.

“Increased estrogen levels in anti-Scl-70 antibody-positive males were associated with an increased risk of death.”

This study, along with others, indicates that high estrogen may play a major role in the severity of autoimmune diseases.

You should always consult a healthcare practitioner about diagnosing and treating any health-related problems.

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Most men have high estrogen — here’s how I fixed mine

A few years ago, on a lab result, my estrogen was through the roof. 

And my doctor didn’t care — he said I should lose weight and take a statin. 

So I started researching everything I could find about estrogen.

Turns out, men who have a bit of a belly almost always have high estrogen.

This leads to “rockiness” problems…

…so after a lot of trial and error and reading, I discovered a simple way to lower my estrogen levels. And I use it constantly.

Best of all, my wife LOVES the “new me” with low estrogen because the difference in my body is SO VISIBLE when we make love.

Click here to see how I lowered my estrogen and improved EVERYTHING including in the bedroom…

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Matt Cook is editor-in-chief of Daily Medical Discoveries. Matt has been a full time health researcher for 26 years. ABC News interviewed Matt on sexual health issues not long ago. Matt is widely quoted on over 1,000,000 websites. He has over 300,000 daily newsletter readers. Daily Medical Discoveries finds hidden, buried or ignored medical studies through the lens of 100 years of proven science. Matt heads up the editorial team of scientists and health researchers. Each discovery is based upon primary studies from peer reviewed science sources following the Daily Medical Discoveries 7 Step Process to ensure accuracy.

 

Estradiol levels are elevated in older men with diffuse cutaneous SSc and are associated with decreased survival

https://arthritis-research.biomedcentral.com/articles/10.1186/s13075-019-1870-6