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Is this it? Have they finally cured it?
—–Important Message—–
Doctors suffer MORE problems with erectile dysfunction than other men…
And the reason is very shocking:
They OVERTREAT themselves medically…
Look: All men lose a little sensitivity as they get older.
But when you MEDICALLY OVERTREAT, the loss of sensitivity gets WORSE.
Then you don’t have morning wood anymore…
And you descend into penile fibrosis, making it very difficult to get erections at all and have great sex again…
So those doctors are making their erection problems WORSE with their overtreatments.
Fortunately, over 21,262 men have found the solution… And a surprising number of these men are medical men themselves!
What doctors and 21,262 men have done to have amazing 30-minute sex any time they want
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This may be the last post about Alzheimer’s
Alzheimer’s disease destroys neurons.
Our brains have nerve cells that receive, process, and transmit information through electrical and chemical signals.
Sure, new neurons can be generated – but, until now, it didn’t seem that triggering the growth of new neurons was beneficial for Alzheimer’s.
This is probably because they also die off quickly.
According to a new study, exercise can increase brain-derived neurotrophic factor (BDNF).
And BDNF protects new neurons.
The experiments show improvement in memory and brain function in animal models of Alzheimer’s.
These researchers carried out their animal studies at the Massachusetts General Hospital and published their results in Science.
A region of the brain called the hippocampus produces messaging cells called neurons.
Some studies have found that the production of these neurons decreases in Alzheimer’s.
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“Human adult hippocampal neurogenesis has been reported to be lowered in Alzheimer’s disease patients as well as in animal models.”
The effect of decreased neurons on Alzheimer’s disease has not been studied enough.
“Evidence supporting a role for adult hippocampal neurogenesis in Alzheimer’s disease has remained sparse and inconclusive.”
This study set out to determine whether altering the rate of neurogenesis could improve Alzheimer’s symptoms.
For one experiment, the scientists created genetically modified mice.
The mice were designed to have a genetic susceptibility to develop Alzheimer’s disease and they were also genetically modified to have increased neurogenesis.
This experiment with genetically modified mice was a failure.
Increasing neurogenesis alone did not protect mice against Alzheimer’s symptoms.
“Inducing adult hippocampal neurogenesis alone conferred minimal to no benefit for improving cognition in Alzheimer’s modified mice.”
In another experiment, the scientists looked at the effect of exercise.
“We then assessed the role of exercise, a known neurogenic stimulus.”
Scientists know that exercise stimulates the production of new neurons in the brain.
But exercise also causes other biochemical changes in the brain.
The scientists wanted to find out if the combination of neurogenesis and other changes to the brain had a beneficial effect.
“We investigated neurogenesis in conjunction with the biochemical changes induced by exercise on Alzheimer’s symptoms in mice.”
The scientists found that exercise led to the right combination of changes in the brain.
“Exercise-induced adult hippocampal neurogenesis improved cognition in Alzheimer’s mice.”
With exercise, the mice performed better on their behavioral tests.
When scientists examined their brains, they found that the mice had fewer of the amyloid plaques that are the physical hallmark of Alzheimer’s.
“Exercise-induced neurogenesis reduced amyloid beta deposits in Alzheimer’s mice.”
A number of other experiments showed that exercise increased levels of brain-derived neurotrophic factor (BDNF).
Exercise was the key factor that allowed the neurons to survive longer.
When the scientists used drugs and genetic modification to increase neurogenesis and BDNF, they saw improvements in the Alzheimer’s type mice even without exercise.
“Inducing neurogenesis genetically and pharmacologically in combination with elevating BDNF levels mimicked the beneficial effects of exercise.”
Physical exercise is critical to brain health in people with Alzheimer’s. Exercise is important for everyone’s brain, actually.
We also know that neurogenesis slows down before Alzheimer’s becomes apparent.
“Adult hippocampal neurogenesis is impaired before the onset of Alzheimer’s disease pathology.”
In the experiment, the animals exercised by running.
Other human and animal experiments show that both cardio and weight-lifting exercises can increase BDNF and neurogenesis.
You don’t need to be running marathons or squatting massive weights to get the brain benefits of exercise.
But you do need to be getting some physical exercise.
You should always consult a healthcare professional about treating and diagnosing health problems.
—-Important Message—-
Harvard study: A new way to sit on the couch and burn 1,800 calories per day
I was excited when I found out about this.
Because, in theory, it meant I could sit around, do nothing, and burn off 1,800 calories of fat every single day.
This got me hitting the books – because I needed to find out how to burn that fat safely…
…so that it would fuel my body’s needs and not get dumped into my arteries as blood fat.
I went through thousands of studies, studies from Harvard, MIT, and Washington University…
And, to my growing excitement, I discovered some superfoods and little-known supplements that keep fat safely in the cells and burn it off.
These superfoods and little-known supplements are inexpensive, too.
I immediately went to work ordering various powders and potions and superfoods and trying various combinations.
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