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Potassium iodide — how to use it and why you want to

Most people know that iodine is used topically for infection.

But potassium iodide is something else entirely, and works in a completely different way — and at MUCH lower doses.

The iodide ion (I⁻), exclusively, is used by neutrophils — a white blood cell — to form hypoiodous acid (HIO).

This is highly effective against microbes, particularly against fungi and yeast.

Iodide’s antifungal/antibacterial mechanism of action has nothing to do with thyroid hormones. 

Potassium iodide has been used for centuries against fungal infections of all types.

This mineral still finds clinical use in Third World Countries, although in the U.S. and Western Europe it’s been abandoned in favor of more profitable treatments.

Yet, biology doesn’t change so quickly over time: iodide is still very effective, despite it being unfashionable among capitalists.

Rational people just want the safest and most efficacious treatments.

They care little about the income of Pfizer and Merck.

The reason why the neutrophil + iodide (I⁻) combination is so effective against fungi is because these cells have myeloperoxidase. 

Myeloperoxidase is a neutrophil-specific enzyme that converts iodide (I⁻) into hypoiodous acid (HIO) and iodine (I₂).

While it’s true that this enzyme can also convert the more common chloride ion (Cl⁻) into hypochlorite (ClO⁻), this one is far less effective against fungi than hypoiodous acid (HIO).

And you’ll know exactly why if you keep reading. 

But first, just have a look at what potassium iodide can do:

A ten year old boy in India presented with a giant tumor.

Sarcoma was considered, yet a biopsy indicated that they were dealing with a fungus in this case: Basidiobolus haptosporus.

‘The organism responds best to potassium iodide.’ ―Thotan

They had simply given him iodide for months until the tumor had disappeared.

The large doses given — 30 ^mg⁄_kg per day — and length of time administered demonstrate its safety.

Potassium iodide is certainly not particular to Basidiobolus haptosporus.

Potassium iodide works equally well against fungi of all types, including Candida albicans, and is still considered the gold standard treatment for sporotrichosis.

Neutrophils are the most important anti-Candida cell type…

And the fact that sodium chloride doesn’t work against fungi is simple proof that it is the hypoiodous acid (HIO⁻) — and not the hypochlorite (ClO⁻) — that is the prime reason for this.

Yet more direct proof for this is available.

It has actually been demonstrated multiple times that the neutrophil + iodide pair is the most effective combination possible:

In a series of studies, Boston scientists showed how essential neutrophils were against Candida albicans.

Other cell types are much less effective: Candida can actually escape engulfment from macrophages.

And even at an unnaturally-high 2:1 ratio, natural killer cells are only capable of inhibiting its growth by 20%.

In addition to antibiotics and glucocorticoids, low blood neutrophil levels is also a major risk factor for candidemia.

Neutropenia versus candidemia risk ratios have been calculated to be as high as 17.4.

And mice genetically engineered to lack myeloperoxidase have increased susceptibility to fungal, yet not bacterial, infection.

‘In previous studies, we noted that Candida hyphae and pseudohyphae could be damaged and probably killed by neutrophils…’ ―Diamond

But specifically, they showed how essential the enzyme myeloperoxidase is towards the end.

Myeloperoxidase is released by white blood cells in the vicinity of Candida albicans and accounts for about 5% of the neutrophil’s total mass.

These doctor-scientists had a few patients with normal neutrophil levels, yet those also lacking myeloperoxidase completely.

Without this enzyme, neutrophils can do little:

Using purified enzymes, sodium chloride, and sodium iodide, they had determined the most effective combinations.

Without myeloperoxidase present: chloride, iodide, and hydrogen peroxide cannot very much affect Candida species.

‘Neutrophils from neither of two subjects with hereditary myeloperoxidase deficiency damaged the hyphae.’ ―Diamond

When myeloperoxidase is added to the solution, either hypoiodous acid (HIO) or hypochlorite is formed (ClO⁻).

Yet of these two species, it is hypoiodous acid that is always found more effective — making iodide an antifungal agent:

This is a consistent finding, and others have reported the same.

In addition to chloride (Cl⁻), myeloperoxidase can also use the thiocyanate ion (SCN⁻).

This forms the analogous hypo- thiocyanite ion (OSCN⁻), which is also microbe-damaging.

However, iodide has consistently been found to be more effective than this species as well.

This makes iodide the most effective substrate of the most effective enzyme against Candida albicans, released by the most effective white blood cell against it.

Now why is the neutrophil + iodide combination so effective against fungi and yeast specifically…

Yet there are no more effective combinations than with chloride or thiocyanate against bacteria?

The answer is simple: fungi & yeast use chitin and β(1➝3)-glucan as structural components of their cellular and hyphal membranes.

Like cellulose, chitin and β(1➝3)-glucan are polysaccharides having β-bonds.

Hypoiodous acid powerfully oxidizes β-polysaccharides, leading to an insoluble product:

This actually forms the basis for a classic test.

The amount of aldehyde groups present on chitin — i.e. the amount already oxidized — can be determined by measuring the amount of hypoiodous acid needed to complete the task.

Although hypochlorite can also oxidize chitin and cellulose, this can only significantly occur at very low pH.

In the human body, at basic pH ≈ 7.4, hypochlorite (ClO⁻) is found mostly as a charged species.

This means that it would be repelled by the negatively-charged hydroxyl groups of chitin.

‘Hypothiocyanite has a pKa of 5.3. It is more acidic than hypohalides that have pK_as of 7.5 (HOCl), 8.6 (HOBr), and 10.6 (HOI). ‘―Bafort

The hypoiodite ⇌ hypoiodous acid equilibrium pair has a much higher pK_a, meaning that it will be found primarily in the neutral acid form at physiological pH.

This fact is crucial for the idiosyncratic effects displayed by hypoiodous acid against fungi:

Neutral molecules can interact with and penetrate cell membranes, while those more negatively-charged — i.e. hypochlorite and hypothiocyanite — are repelled and are to be found more in the extracellular space.

And of the three neutrophil halogen products possible (i.e. ClO⁻, BrO⁻, and HIO⁻), it is the hypoiodite ⇌ hypoiodous acid pair that has the highest oxidizing potential:

‘Hypohalous acids rank as follows, with increasing oxidative strength: OI⁻ < OBr⁻ < OCl⁻.’ ―Bafort

Moreover, the di-iodine (I₂) also produced by myeloperoxidase can iodinate lipids, including the Candida albicans growth factor prostaglandin E₂ and immune evading 3,18-HETE.

Candida albicans synthesizes and emits prostaglandin E₂, which acts to stimulate its yeast ➝ hyphal transition.

It is far more difficult to chlorinate a lipid than iodinate one.

It has been experimentally determined that iodine and hypoiodous acid derived from neutrophils can add to yeast cells, which is correlated with their death:

So even small amounts of potassium iodide, such as from kelp, can help the immune system fight yeast around the clock.

The Japanese consume 1.2 milligrams of iodide per day, on average, largely derived from seaweed.

These milligram-sized intakes are justified by tradition, and its safety is verified by the good health of the Japanese.

Iodide can also be used to make thyroid hormone.

Yet, iodide hyper-accumulates in the thyroid where too much has been shown to cause lipid peroxidation.

This occurs because the thyroid has its own peroxidase enzyme that it uses to make thyroid hormone — i.e. thyroid peroxidase.

For this reason, it’s wise to start slow and incrementally increase the dose.

Although iodide is an essential mineral, taking too much at once after being deficient can shock the thyroid.

‘When 100 μM chloride was substituted for iodide, no significant killing of C. albicans was seen in either the MPG or the Fe²⁺ system with 100 μM H₂0₂.’ ―Levitz

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Matt Cook is editor-in-chief of Daily Medical Discoveries. Matt has been a full time health researcher for 26 years. ABC News interviewed Matt on sexual health issues not long ago. Matt is widely quoted on over 1,000,000 websites. He has over 300,000 daily newsletter readers. Daily Medical Discoveries finds hidden, buried or ignored medical studies through the lens of 100 years of proven science. Matt heads up the editorial team of scientists and health researchers. Each discovery is based upon primary studies from peer reviewed science sources following the Daily Medical Discoveries 7 Step Process to ensure accuracy.