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Is gut inflammation causing ED? Here’s how to fix it fast…
—–Important Message—–
How anyone can have hours of sexual bliss…
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They did… And they recorded everything in ancient books.
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Iron gut = rod of steel
EVERYONE reading this has gut problems. EVERYONE. Sometimes it is called IBS, sometimes it’s called Crohn’s, sometimes it’s called colitis.
Notice that one of these problems is called Crohn’s…
Even though most guys haven’t been diagnosed with Crohn’s, EVERY guy has some inflammation in the gut that causes the same problems.
And it’s possible that we ALL have Crohn’s disease to some degree…
Crohn’s guys just have it worse…
But pay close attention – this is about YOU.
Crohn’s disease is one of a number of inflammatory bowel diseases.
It can create pain and inflammation anywhere throughout the digestive tract.
A major complication with Crohn’s is the inability to maintain a healthy body weight.
Current treatments for gut problems such as Crohn’s are pretty limited.
A number of new discoveries have allowed scientists to alter the bacteria associated with Crohn’s… in mice.
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This has led researchers to believe they may have found a target for treating Crohn’s disease.
A new study conducted by several American universities and published in the journal Science Translational Medicine promises hope for people suffering from Crohn’s disease.
It also offers insights into other inflammatory conditions related to gut bacteria.
Some bacteria within the digestive system can be defined as good and some can be defined as bad.
We all have good gut bacteria and bad gut bacteria.
Gut dysbiosis is a condition where you have an excess of bad bacteria. The paper centers around the problem of gut dysbiosis.
The increase in bad bacteria that leads to gut dysbiosis causes a whole host of problems.
Inflammation and diarrhea are most relevant to Crohn’s disease.
“Gut dysbiosis during inflammatory bowel disease involves alterations in the gut microbiota associated with inflammation of the host gut.”
The scientists examined fecal samples from patients with Crohn’s disease.
They found a link between some types of bacteria, the severity of Crohn’s disease, and the production of certain amino acids.
“We found an association between disease severity, gut dysbiosis, and bacterial production of free amino acids.”
Amino acids are the building blocks of protein. In the case of Crohn’s, they are produced by gut bacteria.
The increase in these amino acids indicates an increase in the amount of certain strains of bacteria.
The scientists then studied nitrogen metabolism in the guts of mice.
This examination revealed an increase in an enzyme called urease.
The increased bacterial urease allowed gut bacteria to produce more of the amino acids previously identified.
“Bacterial urease led to the transfer of nitrogen to the gut microbiota where it was used for amino acid synthesis.”
The urease enzyme, created by gut bacteria, led to the increased synthesis of amino acids.
So the scientists killed off most of the gut bacteria in a group of mice.
Then they infected them with a bacterial population known to produce high amounts of urease.
The infection with high-urease-producing bacteria led to downstream changes in the bacterial colony.
Urease-producing bacteria create food for other bacteria.
Creating a high-urease bacterial population in the mouse gut led to an increase in proteobacteria species.
“Inoculation with E. coli engineered to express urease led to dysbiosis of the gut microbiota, resulting in a predominance of Proteobacteria species.”
The study found that in these mice (engineered to express high urease) inflammation in their gastrointestinal systems increased.
They showed that urease-producing bacteria could cause symptoms of Crohn’s disease.
“This was associated with a worsening of immune-mediated colitis in these animals.”
This discovery shows that when gut dysbiosis leads to increased bacterial urease, this can lead to downstream inflammation from proteobacteria fed by urease.
Because of this discovery, scientists can begin to design ways to target urease-producing bacteria…
Therefore they can target the proteobacteria that feed on the products of urease and are associated with colitis and Crohn’s.
One way to do this would be to create a bacterial population that naturally produces lower levels of urease.
Once this bacterial population is identified in animal models, it can possibly be used in humans.
It is possible to kill off enough bacteria with antibiotics and propylene glycol to alter the gut bacteria population.
This antibacterial and repopulation method has already been proven to alter internal levels of urease created by gut bacteria.
These are exciting times for sufferers of Crohn’s disease, other inflammatory bowel diseases, and many other diseases associated with intestinal dysbiosis.
You should seek medical advice about inflammatory bowel disease or any similar health problems.
—–Important Message—–
Gut problems? Fixing them raises testosterone…
Here’s how gut problems can lead to erection problems:
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