Liver cancer is increasing rapidly.
What is also rapidly increasing is liver function problems.
So if you have a lab test that reveals elevated liver enzymes, today’s newsletter is for you.
Or if you just want to know how to prevent liver problems so that you can live longer and be healthier.
How big of a problem do you think liver issues are today?
Here is a chart showing the increase in liver cancer that’s been going on.
So what is the cause of liver cancer?
The liver’s primary function is to be the chemical factory for the body.
The liver makes numerous chemicals and hormones and compounds for the rest of the body.
But the liver has another function as well.
The liver is the police, court, and executioner for the vast array of toxins that we eat in our foods every day.
The liver has to process these toxins and get rid of them.
It does this either by turning them into something harmless, peeing them out, or defecating them out.
It also deposits the toxins on our skin so that we can shed them.
So peeing, defecating, are shedding are the primary tools the liver has to deal with all the toxins that we consume.
There are two primary types of toxins.
First, we have endotoxins.
Endotoxins are produced by numerous bacteria in our gut.
The most harmful endotoxin is known as LPS, lipopolysaccharide.
The liver has to deal constantly with high levels of LPS from our food.
Another source of toxins are the toxins that are found in moldy foods.
These are found in any foods that were moldy — or even cow milk or cheese that was moldy.
Much mold produces aflatoxin.
The aflatoxin is the most common toxin made by molds — but, there are many other toxins made by molds.
Some molds produce good things too, such as the mold in blue cheese.
But most mold is very, very harmful.
It turns out that when you combine mold aflatoxin with endotoxins, it’s a much bigger challenge for the liver.
The combination is a lot of HARD work for the liver.
They seem to work effectively as a deadly combination.
The aflatoxin & endotoxin mix really hurts the liver.
In fact, it’s probably contributing to the increase in liver cancer.
In this study, researchers gave rats endotoxins combined with aflatoxin.
They determined that the combination is highly toxic to the human body — specifically to the liver.
They also found that there are some things that may help reduce the harm of this damaging duo.
Aspirin may reduce the effects of these toxins
Our studies reveal that aspirin prevents TNF-alpHa and IL-1-induced NKFB activation in a dose-dependent manner.
This study is one of many that shows how aspirin can reduce the effects of TNF-Alpha.
TNF-Alpha is one of the mechanisms that is triggered by endotoxins and aflatoxins.
This is signaling protein that indicates inflammation in the body.
Raise your protein levels!
In this next study, they found that higher protein levels vastly reduce the damage to the liver that is created by endotoxins and aflatoxins.
Rats that were a fed higher protein diet had radically less liver damage than those fed a lower protein diet.
One of the many mistakes that people are making today is not consuming enough protein.
Protein helps reduce the damage to the liver that is caused by the constant barrage of endotoxins and aflatoxins
We are exposed to this combination every single day.
And something as simple as eating enough protein can protect us.
In these studies, higher protein supports the liver function.
Higher protein consumption is vitally important in preventing liver cancer.
And it’s crucial to supporting the liver and its many functions.
There is nothing better than higher protein to help the body remove toxins.
Citations
Liver and intrahepatic bile duct (cancer.org)
https://cancerstatisticscenter.cancer.org/_ga=1.20775741.42002471.1462815277#/cancer-site/Liver%20and%20intrahepatic%20bile%20duct
Bacterial lipopolysaccharide enhances aflatoxin B1 hepatotoxicity in rats by a mechanism that depends on tumor necrosis factor α
http://onlinelibrary.wiley.com/doi/10.1053/jhep.2001.20643/full
Aspirin inhibits TNFalpha- and IL-1-induced NF-kappaB activation and sensitizes HeLa cells to apoptosis.
http://www.ncbi.nlm.nih.gov/pubmed/15036249
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