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There’s even a start-up company dedicated to this new injection…
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Here’s why men crave this new anti-FLT3 injection.
Peripheral neuropathic pain is a chronic illness affecting up to 10% of the population.
The pain is caused by over-sensitization of neurons.
Currently, there is no effective treatment.
But scientists in France believe they have found a mechanism to block peripheral neuropathic pain at the source.
Scientists at the Institute for Neurosciences of Montpellier and the Laboratory for Therapeutic Innovation in Strasbourg conducted these animal studies. They published the results in the journal Nature Communications.
In the experiment, the researchers injured mice in order to create peripheral neuropathic pain.
10 days later, the scientists examined the nerves of the injured animals – looking for chemical inflammatory markers.
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Scientists found an elevated amount of an inflammatory marker known as FL.
“We found intense FL activity in the nerve at the site of injury.”
The researchers did not see this increase in FL in animals that were not subjected to the procedure that creates peripheral neuropathic pain.
So the scientists wanted to see if FL alone (without actual injury) could create a neuropathic pain response in the mice.
“To assess whether FL could generate peripheral neuropathic pain symptoms, uninjured mice were injected with FL.”
The mice injected with the inflammatory marker FL indeed displayed symptoms of neuropathic pain.
“A single FL injection induced a dose-dependent increase in the percentage of paw withdrawal, a hallmark of peripheral neuropathic pain.”
After getting an injection of FL, the animals were jumpy to the touch.
Increasingly, they withdrew their paws from contact with a testing device – showing they were experiencing more pain.
The exaggerated pain response was present in the animals for a long time after being injected with FL.
“The neuropathic pain response to FL was present 5 hours post-injection and persisted for at least 2 days.”
Injections of FL also made mice hypersensitive to hot and cold – further indicating neuropathic pain.
“FL-injected mice also displayed thermal hypersensitivity in response to cold and hot stimuli.”
The scientists wanted to backtest these findings.
To do this, they created mutant mice that had a reduced number of receptors for the inflammatory FL.
Mice with fewer receptors for FL did not show signs of pain hypersensitivity after being injected with FL.
“Mutant mice without FLT3 receptors failed to develop pain hypersensitivity following FL injection.”
(FLT3 refers to FL receptors.)
These results show that FL affects the FLT3 receptor and this effect is a cause of peripheral neuropathic pain.
“This indicates that FL-induced hypersensitivity resulted from activation of its FLT3 receptor.”
The study also looked at changes in the sensory neurons – these changes in cells are related to peripheral neuropathic pain.
Injection of FL created the same changes to the sensory neurons that occur in peripheral neuropathic pain caused by nerve injury.
“FL causes changes in sensory neurons that are normally caused by peripheral nerve injury.”
It may be that nerve injury increases FL, and then FL activates the FLT3 receptors – leading to peripheral neuropathic pain.
If we can interrupt FL or FLT3, then perhaps we can put an end to peripheral neuropathic pain.
The mutant FLT3 mice that were immune to peripheral neuropathic pain did not show any other abnormal behaviors.
They responded to normal pain in the same way that standard lab mice do.
In another stage of the experiment, the scientists developed an injection that could downregulate FLT3 receptors in normal mice.
In theory, this could alleviate neuropathic pain.
First, they gave the mice FL. That created peripheral neuropathic pain.
Then they gave the mice the injection to downregulate the FL receptor (FLT3).
“To see if peripheral neuropathic pain could be reversed by FLT3 downregulation, we injected FLT3 down-regulator into the mice.”
The anti-FLT3 injection worked.
These scientists have figured out how to reverse peripheral neuropathic pain.
“The injection effectively down-regulated FLT3 expression and mechanical hypersensitivity without affecting normal mechanical pain sensation.”
The scientists then tested a safer drug version of the anti-FLT3 injection.
Scientists found that injection with the drug version of anti-FLT3 (BDT001) also reversed neuropathic pain.
“After systemic administration of BDT001 in mice, FL-induced neuropathic pain was completely abrogated.”
This promising technique needs to be safety tested in humans.
Currently, a start-up company is working on furthering these findings.
You should see a healthcare professional about diagnosing and treating pain.
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