I tried these words on 5 women and this happened

I’ve never seen a woman get so wet…Just 3 words… 3 words get her feeling a sudden intensity…

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—-Important Message From Lawrence Lanoff—-

These 3 words trigger her animal side

Just 3 words… 3 words get her feeling a sudden intensity…

…a dampness between her thighs…

…an excitement that ripples through her belly and turns to warmth just below…

With this specific naughty word, you can access her deepest, most secret sexual desires…

Just whisper these 3 words into her ear tonight…

———-

This can give you iron-clad immunities to most illnesses

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Immunity from disease is a hot topic right now – but immunity is always important.

Every day we are bombarded with disease-causing bacteria and viruses.

Some people may notice no effects from these infectious agents.

But for other people, the same pathogen could cause severe disease and even death.

The reasons for these differing reactions to infectious agents are differences in how the immune system performs.

You’re probably aware that for most infectious diseases children have a very high immunity…

…while older people are in the highest risk category.

The thymus gland in the chest produces many of the particles we use to kill infections.

As we age, the thymus gland tends to degenerate – it’s a large reason why older people tend to have poor immunity.

But it’s not simply time that causes damage to the thymus and weakens our immune system.

Stress hormones and nutrient deficiencies cause artificial aging and poke in our immune system defenses…

…but these problems can be fixed.

For example – the stress hormone cortisol causes changes to the thymus gland similar to that seen in aging.

Cortisol tends to go up with age too. And by lowering cortisol we can recharge our immune systems.

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These animal experiments were carried out at University College Cardiff in the United Kingdom. The results were published in Age and Ageing.

In humans and animals there are major changes to the thymus gland seen in aging.

“The thymus gland of old mice is quite different from that seen in young animals. The gland was much smaller in older animals – 80% weight loss.”

This has severe implications for immunity – because the thymus gland produces many of the T cells that we used to fight disease.

Scouting cells in the blood look for infectious viruses and bacteria.

Once they find them they relate that information to the thymus gland.

Then the thymus gland creates immune factors which seek and destroy these infectious agents.

So a weakened thymus gland means a weakened immune system.

But age alone does not determine the health of the thymus.

(I previously reported on the effect of zinc in recharging the “aged” thymus.)

Cortisol is a stress hormone produced by humans every day. A little cortisol is essential and without some we would die.

But cortisol has negative effects too. And chronic elevations of cortisol cause “aging”.

If you see identical twins where one looks much older than the other, you can bet that that twin has had higher levels of cortisol over their lifetime.

So this research was designed to see the effect of cortisol on the thymus glands in mice.

Researchers gave lab mice high doses of cortisol and looked to see if there are any changes to the function of this immune cell factory.

Within two days cortisol triggered a massive drop in killer cells – immune cells which kill infectious disease and fight cancer.

“Cortisol treatment resulted, within two days, in a loss of killer cells.”

Instead, the thymus started producing more fibroblasts – generally needed for wound healing…

…but in excess they can cause tissue and blood vessel stiffness (AKA aging).

The cortisol treated glands also ramped up the production of macrophages…

…immune cells which are generally less useful than killer cells.

“Cortisol treatment resulted in a rise in the proportion of macrophages and fibroblasts.”

The effects of cortisol were almost identical to the effects of aging on the thymus gland.

“This pattern was similar to that characteristic of the old thymus.”

High cortisol is one of the major causes of accelerated aging – and it’s very important to control this stress hormone.

Many people can significantly lower their cortisol levels using a few simple tips and tricks.

—-Important Message About Lowering Cortisol… with Sugar?—-

Do you know what eventually happens to men who only burn fat and not sugar?

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When your body is burning fat, your body is in what I call emergency mode.

And there are many bad stress hormones that are produced when your body is burning fat in emergency mode.

And what I mean by emergency mode is…

  • High estrogen
  • High cortisol
  • High serotonin

These are all stress hormones that in high levels result in massive inflammation throughout the body.

Which can turn into diabetes, high blood pressure, acid reflux, prostate inflammation, sometimes even cancer.

And it almost always shows up in men in the form of erections problems.

Fat burning emergency mode just sucks. It’s awful for your health.

It’s awful for your brain. And it’s awful for your sex life.

So how do you stop it?

If you want to feel young and healthy like a kid again, you must burn SUGAR, not fat.

And I’ve come up with a super simple “sippy cup” activity that kick starts the body into burning sugar again.

Try it here

———-


Matt Cook is editor-in-chief of Daily Medical Discoveries. Matt has been a full time health researcher for 26 years. ABC News interviewed Matt on sexual health issues not long ago. Matt is widely quoted on over 1,000,000 websites. He has over 300,000 daily newsletter readers. Daily Medical Discoveries finds hidden, buried or ignored medical studies through the lens of 100 years of proven science. Matt heads up the editorial team of scientists and health researchers. Each discovery is based upon primary studies from peer reviewed science sources following the Daily Medical Discoveries 7 Step Process to ensure accuracy.
The cellular composition of thymus: a comparison between cortisol-treated and aged C57-BL micehttps://pubmed.ncbi.nlm.nih.gov/4669870/